I graduated with a Bachelor’s degree in Pharmacy from the University of Baghdad in 2013 and subsequently worked at the College of Pharmacy at the same university. I then pursued a Master's degree in the Department of Pharmacology and Toxicology at Baghdad University,After completing my degree in 2022, I returned to the College of Pharmacy at the University of Baghdad to work as an Assistant Lecturer.
My research interest lies in the field of pharmacology with a specific focus on toxicity and its management. I am particularly interested in understanding the mechanisms through which various pharmacological agents exert toxic effects on biological systems.
M.Sc. Pharmacology and Toxicology (2022), College of Pharmacy, University of Baghdad
B.Sc. Pharmacy (2013), College of Pharmacy, University of Baghdad
Practical Organic Chemistry II Practical Organic Pharmaceutical Chemistry II Practical Physiology I Practical Clinical Toxicology II Practical Pharmacology II
Inflammatory bowel disease (IBD) is a common chronic inflammatory disease of the gastrointestinal tract, including ulcerative colitis and Crohn's disease. ulcerative colitis (UC) disease is characterized by chronic, persistent, recurrent, and nonspecific intestinal ulcers and mucosal inflammation. This study investigated the protective effects of cinnamic acid on dextran sodium sulfate (DSS) induced ulcerative colitis in mice. Forty adult male mice were collected and randomly divided into five groups, group Ӏ received a suspension of distill water and poloxamer, and group ӀӀ received 3% DSS in drinking water for 7 consecutive days. Two treatment groups received an oral suspension of cinnamic acid 50 and 25 mg/kg respectively an
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Objective: To study the protective eff ects of cinnamic acid on dextran sodium sulfate (DSS) induced ulcerative colitis (UC) in mice. Materials and methods. Forty adult male mice were randomLy divided into fi ve groups, control group, an induction group received 3% DSS in drinking water for 7 consecutive days. Two treatment groups received oral suspension of cinnamic acid 50 and 25 mg/kg, respectively and 3% DSS in drinking water, for 7 consecutive days. The fi nal group received oral suspension of cinnamic acid 50 mg/kg for the latter 7 days without DSS in drinking water. All the animals were euthanized on day eight. The colon of animals was extracted and divided into two sections, the middle was homogenized and biochemically analy
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